<?xml version="1.0" encoding="UTF-8"?>
<collection>
<dc:dc xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:invenio="http://invenio-software.org/elements/1.0" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd"><dc:identifier>doi:10.1093/cvr/cvac066.022</dc:identifier><dc:language>eng</dc:language><dc:creator>Pérez Martínez, A.</dc:creator><dc:creator>García Mendívil, L.</dc:creator><dc:creator>López Yus, M.</dc:creator><dc:creator>Mancebón Sierra, F. J.</dc:creator><dc:creator>Vaca Núñez, A.S.</dc:creator><dc:creator>Ballester Cuenca, C.</dc:creator><dc:creator>Pérez Zabalza, M.</dc:creator><dc:creator>Kohler, R.</dc:creator><dc:creator>Pueyo, E.</dc:creator><dc:creator>Oliván Viguera, A.</dc:creator><dc:title>SK channels contribution to ventricular electrophysiology in heart failure patients</dc:title><dc:identifier>ART-2022-129658</dc:identifier><dc:description>Heart failure (HF) is characterized by deterioration of the electrical and contractile function of the heart due to structural and functional remodelling, leading to development of arrhythmias and increased sudden cardiac death risk. SK channels are a type of calcium-activated potassium channels that do not play a relevant role in normal ventricular electrophysiology. However, it has been hypothesized that these channels become more relevant in pathologies such as HF. Nontheless, their role in human ventricular electrophysiology is not fully characterized.</dc:description><dc:date>2022</dc:date><dc:source>http://zaguan.unizar.es/record/118729</dc:source><dc:doi>10.1093/cvr/cvac066.022</dc:doi><dc:identifier>http://zaguan.unizar.es/record/118729</dc:identifier><dc:identifier>oai:zaguan.unizar.es:118729</dc:identifier><dc:identifier.citation>Cardiovascular research 118, Supp. 1 (2022), [1 pp.]</dc:identifier.citation><dc:rights>All rights reserved</dc:rights><dc:rights>http://www.europeana.eu/rights/rr-f/</dc:rights><dc:rights>info:eu-repo/semantics/openAccess</dc:rights></dc:dc>

</collection>