000170098 001__ 170098
000170098 005__ 20260318155254.0
000170098 0247_ $$2doi$$a10.1016/j.endien.2022.03.006
000170098 0248_ $$2sideral$$a148537
000170098 037__ $$aART-2022-148537
000170098 041__ $$aeng
000170098 100__ $$aGarcía Ventura, María
000170098 245__ $$aPerinatal factors influence on the neurocognitive development of children born small for gestational age (SGA) during the first 2 years of life
000170098 260__ $$c2022
000170098 5203_ $$aBackground and objective: Children born small for gestational age (SGA) show higher risk of neurodevelopmental and cognitive abnormalities. The objective of this study is to determine in children born SGA the neurodevelopment during the first 2 years of life and to establish the influence of anthropometric data, gestational age, multiple gestation and perinatal factors.
Patients and method: Observational, prospective, descriptive and analytical study of the neurocognitive assessment performed, with Brunet-Lézine test, on SGA children (n = 91) from 3 to 24 months of age, comparing with own controls.
Results: Ninety-one SGA children, 47% girls, 83.5% single pregnancies; mean gestational age 37.7 weeks (standard deviation (SD) 2.1). Weight at birth 2053 g (SD 433.1), length 43.9 cm (SD 2.6) and head circumference 31.7 cm (SD 1.7). The SGA population shows significantly lower neurodevelopment than the control population, with a tendency to improve during the first 2 years of life. There are no differences by sex. SGA children born to multiple gestations have lower neurodevelopment only during the first year of life. There is a direct and positive correlation between weight, length and head circumference with neurocognitive development at 6, 9, 12 and 18 months. Gestational age correlated with better neurodevelopment at 3 and 6 months.
Conclusions: Children born SGA present lower neurodevelopment than the control population. A greater impact on weight, length, and head circumference at birth is correlated with poorer neurocognitive development. Multiparity does not show significant influence on neurodevelopment evolution.
000170098 540__ $$9info:eu-repo/semantics/closedAccess$$aAll rights reserved$$uhttp://www.europeana.eu/rights/rr-f/
000170098 590__ $$a1.9$$b2022
000170098 591__ $$aNUTRITION & DIETETICS$$b72 / 87 = 0.828$$c2022$$dQ4$$eT3
000170098 591__ $$aENDOCRINOLOGY & METABOLISM$$b128 / 144 = 0.889$$c2022$$dQ4$$eT3
000170098 655_4 $$ainfo:eu-repo/semantics/article$$vinfo:eu-repo/semantics/publishedVersion
000170098 700__ $$0(orcid)0000-0002-2865-5813$$ade Arriba Muñoz, Antonio$$uUniversidad de Zaragoza
000170098 700__ $$aPuga González, Beatriz
000170098 700__ $$aAbenia Usón, Pilar
000170098 700__ $$aSánchez Malo, María José
000170098 700__ $$0(orcid)0000-0003-2832-2266$$aLabarta Aizpún, José Ignacio$$uUniversidad de Zaragoza
000170098 7102_ $$11011$$2670$$aUniversidad de Zaragoza$$bDpto. Microb.Ped.Radio.Sal.Pú.$$cÁrea Pediatría
000170098 773__ $$g69, 4 (2022), 271-278$$pEndocrinol. diabetes nutr. (Engl. ed.)$$tEndocrinología, diabetes y nutrición (English ed.)$$x2530-0180
000170098 8564_ $$s1093655$$uhttps://zaguan.unizar.es/record/170098/files/texto_completo.pdf$$yVersión publicada
000170098 8564_ $$s1898935$$uhttps://zaguan.unizar.es/record/170098/files/texto_completo.jpg?subformat=icon$$xicon$$yVersión publicada
000170098 909CO $$ooai:zaguan.unizar.es:170098$$particulos$$pdriver
000170098 951__ $$a2026-03-18-13:52:15
000170098 980__ $$aARTICLE