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    <subfield code="2">doi</subfield>
    <subfield code="a">10.1016/j.atherosclerosis.2018.06.870</subfield>
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    <subfield code="2">sideral</subfield>
    <subfield code="a">107063</subfield>
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  <datafield tag="037" ind1=" " ind2=" ">
    <subfield code="a">ART-2018-107063</subfield>
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  <datafield tag="041" ind1=" " ind2=" ">
    <subfield code="a">eng</subfield>
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  <datafield tag="100" ind1=" " ind2=" ">
    <subfield code="a">Marco-Benedí, V.</subfield>
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  <datafield tag="245" ind1=" " ind2=" ">
    <subfield code="a">Disappearance of recurrent pancreatitis after splenectomy in familial chylomicronemia syndrome</subfield>
  </datafield>
  <datafield tag="260" ind1=" " ind2=" ">
    <subfield code="c">2018</subfield>
  </datafield>
  <datafield tag="506" ind1="0" ind2=" ">
    <subfield code="a">Access copy available to the general public</subfield>
    <subfield code="f">Unrestricted</subfield>
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  <datafield tag="520" ind1="3" ind2=" ">
    <subfield code="a">Background and aims: Recurrent pancreatitis is a severe complication of familial chylomicronemia syndrome (FCS) mainly secondary to lipoprotein lipase deficiency. The mechanism and interindividual variability of pancreatitis in FCS are not fully understood, but abnormalities in the drainage system of pancreatic veins could be involved. 
Methods and results: Two cases of typical FCS are described with a past history of recurrent pancreatitis that dramatically improved after splenectomy performed in both cases for reasons non-related to FCS. 
Conclusions: These are the first reports of the disappearance of pancreatitis after splenectomy in FCS and they should be considered of anecdotal nature at this time. The disappearance of pancreatitis following splenectomy could be in part due to subsequent improvements in pancreatic drainage. Extrahepatic portal hypertension induced by hypertriglyceridemic splenomegaly leading to pancreatic congestion could also be a contributing factor.</subfield>
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    <subfield code="9">info:eu-repo/grantAgreement/ES/ISCIII-ERDF/CIBERCV-CB16-11-00451</subfield>
    <subfield code="9">info:eu-repo/grantAgreement/ES/ISCIII/FIS/IIS16-0114</subfield>
    <subfield code="9">info:eu-repo/grantAgreement/ES/ISCIII/FIS/PIE16-00022</subfield>
    <subfield code="9">info:eu-repo/grantAgreement/ES/ISCIII/FIS/PI15-01983</subfield>
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    <subfield code="9">info:eu-repo/semantics/openAccess</subfield>
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    <subfield code="u">http://creativecommons.org/licenses/by-nc-nd/3.0/es/</subfield>
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    <subfield code="b">2018</subfield>
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  <datafield tag="591" ind1=" " ind2=" ">
    <subfield code="a">PERIPHERAL VASCULAR DISEASE</subfield>
    <subfield code="b">12 / 65 = 0.185</subfield>
    <subfield code="c">2018</subfield>
    <subfield code="d">Q1</subfield>
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  <datafield tag="591" ind1=" " ind2=" ">
    <subfield code="a">CARDIAC &amp; CARDIOVASCULAR SYSTEMS</subfield>
    <subfield code="b">37 / 136 = 0.272</subfield>
    <subfield code="c">2018</subfield>
    <subfield code="d">Q2</subfield>
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    <subfield code="b">2018</subfield>
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  <datafield tag="593" ind1=" " ind2=" ">
    <subfield code="a">Cardiology and Cardiovascular Medicine</subfield>
    <subfield code="c">2018</subfield>
    <subfield code="d">Q1</subfield>
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  <datafield tag="700" ind1=" " ind2=" ">
    <subfield code="a">Lamiquiz-Moneo, I.</subfield>
    <subfield code="0">(orcid)0000-0002-9647-0108</subfield>
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  <datafield tag="700" ind1=" " ind2=" ">
    <subfield code="a">Álvarez-Sala, L.A.</subfield>
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  <datafield tag="700" ind1=" " ind2=" ">
    <subfield code="a">Civeira, F.</subfield>
    <subfield code="u">Universidad de Zaragoza</subfield>
    <subfield code="0">(orcid)0000-0001-7043-0952</subfield>
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    <subfield code="1">1007</subfield>
    <subfield code="2">610</subfield>
    <subfield code="a">Universidad de Zaragoza</subfield>
    <subfield code="b">Dpto. Medicina, Psiqu. y Derm.</subfield>
    <subfield code="c">Area Medicina</subfield>
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  <datafield tag="773" ind1=" " ind2=" ">
    <subfield code="g">275 (2018), 342-345</subfield>
    <subfield code="p">Atherosclerosis</subfield>
    <subfield code="t">Atherosclerosis</subfield>
    <subfield code="x">0021-9150</subfield>
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