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  <datafield tag="100" ind1=" " ind2=" ">
    <subfield code="a">del Buey, María A.</subfield>
    <subfield code="u">Universidad de Zaragoza</subfield>
    <subfield code="0">(orcid)0000-0002-9325-9964</subfield>
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  <datafield tag="245" ind1=" " ind2=" ">
    <subfield code="a">An Update on Corneal Biomechanics and Architecture in Diabetes</subfield>
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    <subfield code="c">2019</subfield>
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    <subfield code="a">In the last decade, we have witnessed substantial progress in our understanding of corneal biomechanics and architecture. It is well known that diabetes is a systemic metabolic disease that causes chronic progressive damage in the main organs of the human body, including the eyeball. Although the main and most widely recognized ocular effect of diabetes is on the retina, the structure of the cornea (the outermost and transparent tissue of the eye) can also be affected by the poor glycemic control characterizing diabetes.  e different corneal structures (epithelium, stroma, and endothelium) are affected by specific complications of diabetes.  e development of new noninvasive diagnostic technologies has provided a better understanding of corneal tissue modifications.  e objective of this review is to describe the advances in the knowledge of the corneal alterations that diabetes can induce</subfield>
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    <subfield code="a">Casas, Paula</subfield>
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    <subfield code="a">de la Rica, Marisa</subfield>
    <subfield code="u">Universidad de Zaragoza</subfield>
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    <subfield code="a">Subirón, Ana B.</subfield>
    <subfield code="u">Universidad de Zaragoza</subfield>
    <subfield code="0">(orcid)0000-0002-5676-4179</subfield>
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    <subfield code="a">Lanchares, Elena</subfield>
    <subfield code="u">Universidad de Zaragoza</subfield>
    <subfield code="0">(orcid)0000-0002-2959-0628</subfield>
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    <subfield code="a">Ascaso, Francisco J.</subfield>
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    <subfield code="a">Universidad de Zaragoza</subfield>
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    <subfield code="p">J. Ophthalmol.</subfield>
    <subfield code="t">Journal of Ophthalmology</subfield>
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