000086992 001__ 86992
000086992 005__ 20200716101519.0
000086992 0247_ $$2doi$$a10.1016/j.jbiomech.2018.12.047
000086992 0248_ $$2sideral$$a110437
000086992 037__ $$aART-2019-110437
000086992 041__ $$aeng
000086992 100__ $$0(orcid)0000-0002-4785-8982$$aCifuentes-De la Portilla, C.
000086992 245__ $$aAnalysis of the main passive soft tissues associated with adult acquired flatfoot deformity development: A computational modeling approach
000086992 260__ $$c2019
000086992 5060_ $$aAccess copy available to the general public$$fUnrestricted
000086992 5203_ $$aAdult acquired flatfoot deformity (AAFD) is a pathology with a wide range of treatment options. Physicians decide the best treatment based on their experience, so the process is entirely subjective. A better understanding of soft tissue stress and its contribution in supporting the plantar arch could help to guide the clinical decision. Traditional experimental trials cannot consistently evaluate the contribution of each tissue. Therefore, in this research a 3-Dimensional FE foot model was reconstructed from a normal patient in order to measure the stress of the passive stabilizers of the arch, and its variation in different scenarios related with intermediate stages of AAFD development. All bones, the plantar fascia (PF), cartilages, plantar ligaments and the spring ligament (SL) were included, respecting their anatomical distribution and biomechanical characteristics. An AAFD evaluation scenario was simulated. The relative contribution of each tissue was obtained comparing each result with a normal case. The results show that PF is the main tissue that prevents the arch elongation, while SL mainly reduces the foot pronation. Long and short plantar ligaments play a secondary role in this process. The stress increment on both PF and SL when one of two fails suggests that these tissues complement each other. These findings support the theory that regards the tibialis posterior tendon as a secondary actor in the arch maintenance, compared with the PF and the SL, because this tendon is overstretched by the hindfoot pronation around the talonavicular joint. This approach could help to improve the understanding of AAFD.
000086992 536__ $$9info:eu-repo/grantAgreement/ES/MINECO/DPI2016-77016-R
000086992 540__ $$9info:eu-repo/semantics/openAccess$$aby-nc-nd$$uhttp://creativecommons.org/licenses/by-nc-nd/3.0/es/
000086992 590__ $$a2.32$$b2019
000086992 591__ $$aENGINEERING, BIOMEDICAL$$b48 / 87 = 0.552$$c2019$$dQ3$$eT2
000086992 591__ $$aBIOPHYSICS$$b38 / 71 = 0.535$$c2019$$dQ3$$eT2
000086992 592__ $$a1.011$$b2019
000086992 593__ $$aBiomedical Engineering$$c2019$$dQ1
000086992 593__ $$aBiophysics$$c2019$$dQ1
000086992 593__ $$aOrthopedics and Sports Medicine$$c2019$$dQ1
000086992 593__ $$aRehabilitation$$c2019$$dQ1
000086992 593__ $$aSports Science$$c2019$$dQ2
000086992 655_4 $$ainfo:eu-repo/semantics/article$$vinfo:eu-repo/semantics/acceptedVersion
000086992 700__ $$aLarrainzar-Garijo, R.
000086992 700__ $$0(orcid)0000-0001-5505-6974$$aBayod, J.$$uUniversidad de Zaragoza
000086992 7102_ $$15004$$2605$$aUniversidad de Zaragoza$$bDpto. Ingeniería Mecánica$$cÁrea Mec.Med.Cont. y Teor.Est.
000086992 773__ $$g84, 14 (2019), 183-190$$pJ. biomech.$$tJournal of Biomechanics$$x0021-9290
000086992 8564_ $$s466280$$uhttps://zaguan.unizar.es/record/86992/files/texto_completo.pdf$$yPostprint
000086992 8564_ $$s34442$$uhttps://zaguan.unizar.es/record/86992/files/texto_completo.jpg?subformat=icon$$xicon$$yPostprint
000086992 909CO $$ooai:zaguan.unizar.es:86992$$particulos$$pdriver
000086992 951__ $$a2020-07-16-09:25:47
000086992 980__ $$aARTICLE